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  • 收录类型=SCI-EXPANDEDx
  • 人物=刘雪松x

5 条 记 录,以下是 1-5

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1Norcantharidin combined with diamminedichloroplatinum inhibits tumor growth and cancerometastasis of hepatic carcinoma in murine被引量:3收藏 分享
作者:Zhang, Xiao-Ping Luo, Lu-Lu Liu, Yong-Qi Liu, Xue-Song An, Fang-Yu
机构:Gansu Univ Chinese Med;Gansu Univ Chinese Med;Gansu Univ Chinese Med
来源:JOURNAL OF CANCER RESEARCH AND THERAPEUTICS  2018年14卷12期
JCR:四分区
影响因子:1.392
关键词:Diamminedichloroplatinum   hepatic carcinoma   norcantharidin  
2Assessment of heavy metal content in cosmetics of Chinese market被引量:1收藏 分享
作者:Liu, Xuesong Zhang, Haoling Zhang, Haolong Jiang, Yuhang Lv, Li
机构:Gansu Univ Tradit Chinese Med
来源:BASIC & CLINICAL PHARMACOLOGY & TOXICOLOGY  2021年128卷
JCR:二分区
影响因子:3.688
3Microbial metabolites in tumor epigenetic regulation被引量:0收藏 分享
作者:Zhang, Wangzheqi Zhang, Haoling Liao, Yan Jiang, Shuya Zhang, Haolong
机构:Naval Med Univ;Naval Med Univ;Univ Sains Malaysia;Naval Med Univ;Henan Med Univ
来源:IMETA  2026年5卷1期
关键词:bidirectional feedback loop   epigenetic modulation   microbial metabolites   precision oncology   tumor epigenetics   tumor microenvironment  
4ATP-P2X7R-NLRP3 Axis as a Unifying Mechanism of ATP-Induced Cell Death in Degenerative Bone and Cardiovascular Diseases被引量:0收藏 分享
作者:Zhang, Jingjing Li, Linzhu Guo, Qianwen Liu, Xuesong Qiao, Qian
机构:Kunming Med Univ;Yunnan Prov Cardiovasc Clin Med Ctr;Yunnan Prov Cardiovasc Clin Med Res Ctr;Gansu Univ Chinese Med;Gansu Univ Chinese Med
来源:JOURNAL OF INFLAMMATION RESEARCH  2026年19卷
JCR:三分区
影响因子:4.631
关键词:ATP-induced cell death   purinergic signaling   mitochondrial dysfunction   NLRP3 inflammasome   degenerative bone disorders   cardiovascular remodeling  
5The complex relationship between oxidative stress and aging: new perspectives on similarities and differences被引量:0收藏 分享
作者:Liu, Xue-song Zhu, Chenglong Zhang, Haoling Zhang, Wangzheqi
机构:Gansu Univ Chinese Med;Naval Med Univ;Univ Sains Malaysia;Naval Med Univ
来源:CRITICAL REVIEWS IN CLINICAL LABORATORY SCIENCES  2026年
JCR:一分区
影响因子:7.721
关键词:Oxidative stress   aging   reactive oxygen species   cellular senescence   mitochondrial dysfunction  
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